Publications

Asano S, Bratz IN, Berwick ZC, Fancher IS, Tune JD, Dick GM. Penitrem A as a tool to understand the role of BK channels in vascular function. J Pharmacol Exp Ther. 2012 Aug;342(2):453-60.

Fancher IS, Dick GM, Hollander JM. Diabetes reduces the function and expression of ATPdependent K+ channels in cardiac mitochondria. Life Sci. 2013 Mar 28;92(11):664-8.

*Rottgen TS, *Fancher IS, Asano S, Widlanski TS, Dick GM. Bisphenol A activates BK channels through effects on α and β1 subunits. Channels. 2014 Jan 29;8(3), 1-9.
*Authors contributed equally.

Fancher IS, Butcher JT, Brooks SD, Rottgen TS, Skaff PR, Frisbee JC, and Dick GM. DPO-1-sensitive K+ channels contribute to the vascular tone and reactivity of resistance arteries from brain and skeletal muscle. Microcirculation. 2015 May;22(4):315-25.

DelloStritto D, Connell PJ, Dick GM, Fancher IS, Klarich B, Fahmy JN, Kang PT, Chen YR, Damron DS, Thodeti CK, and Bratz IN. Differential regulation of TRPV1 channels by H2O2: implications for diabetic microvascular dysfunction. Basic Res Cardiol. 2016 Mar;111(2):21.

Ahn SJ, Fancher IS, Bian JT, Zhang CX, Schwab S, Gaffin R, Phillips SA, and Levitan I. Inwardly-rectifying K+ channels are major contributors to flow-induced vasodilation in resistance arteries. J Physiol. 2017 April 1; 595(7):2339-2364.

Robinson AR, Fancher IS, Sudhahar V, Bian JT, Cook MD, Ushio-Fukai M, Brown MD, Fukai T, and Phillips SA. Short-term Regular Aerobic Exercise Reduces Oxidative Stress Produced by Acute High Intraluminal Pressure in the Adipose Microvasculature. AJP Heart and Circ. 2017 May 1; 312(5):H896-H906.

Ozemek C, Phillips SA, Popovic D, Laddu-Patel D, Fancher IS, Arena R, Lavie CJ. Nonpharmacologic management of hypertension: a multidisciplinary approach. Curr Opin Cardiol. 2017 Jul;32(4):381-388.

*Zinkevich NS, *Fancher IS, Gutterman DG, Phillips SA. Roles of NADPH-oxidase and mitochondria in flow-induced vasodilation of human adipose arterioles: ROS induced ROS release in coronary artery disease. Microcirculation. 2017 Aug;24(6). DOI: 10.1111/micc.12380
*Authors contributed equally.

Robinson AT, Fancher IS, Mahumoud AM, Phillips SA. Microvascular Vasodilator Plasticity Following Acute Exercise. Exerc Sport Sci Rev. 2018 Jan;46(1):48-55.

LeMaster E, Huang RT, Zhang C, Bogachkov Y, Coles C, Shentu TP, Sheng Y, Fancher IS, Ng C, Christoforidis T, Subbaiah PV, Berdyshev E, Qain Z, Eddington D, Lee J, Cho M, Fang Y, Minshall RD, and Levitan I. Proatherogenic Flow Increases Endothelial Stiffness via Enhanced CD36 (Cluster of Differentiation 36)-Mediated OxLDLD (Oxidized Low-Density Lipoprotein) Upatke. Aterieoscler Thromb Vasc Biol. 2018 Jan;38(1):64-75.

†Fancher IS, Ahn SJ, Adamos C, Osborn C, Oh MJ, Fang Y, Reardon CA, Getz GS, Phillips SA, †Levitan I. Hypercholesterolemia-Induced Loss of Flow-Induced Vasodilation and Lesion Formation in Apolioprotein E-Deficient Mice Critically Depend on Inwardly Rectifying K+
Channels. J Am Heart Assoc. 2018 Mar 3;7(5). Pii: e007430. doi: 10.1161/JAHA.117.007430.
†Corresponding authors. Received Editorial in JAHA entitled “Regulation of Kir2.1 Function Under Shear stress and Cholesterol Loading.”

LeMaster E, Fancher IS, Lee J, and Levitan I. Comparative analysis of endothelial cell and subendothelial cell elastic moduli in young and aged mice: Role of CD36. J BioMech. 2018 Jul 25;76:263-268.

Fancher IS, Rubenstein I, Levitan I. Potential strategies to reduce blood pressure in treatment resistant hypertension using FDA-approved nanodrug delivery platforms. Hypertension. 2019 Feb;73(2):250-257.

Boriushkin E, Fancher IS, and Levitan I. Shear-stress sensitive inwardly-rectifying K+ channels regulate developmental retinal angiogenesis by vessel regression. Cell Physiol Biochem. 2019;52(6):1569-1583.

†Fancher IS and Levitan I. Electrophysiological recordings of single-cell ion currents under well-defined shear stress. J Vis Exp. (150), e59776, doi:10.3791/59776 (2019).
†Corresponding author.